1. Immunobiology and immunopathology ofhuman gut mucosa: humoral immunity and intraepithelial LPL are clearly distinguished we conclude that 'true' IEL can mediate this function. We suggest that the documented cytotoxicity is mediated by T cells, as CD3, CD2, and phytohaemagluttinin are similarly effective. The time course of the toxic activity of IEL seems to be longer;Brandtzaeg, P.; Halstensen, T.S.; Kett, K.; Kraici, P.; Kvale, D.; Rognum, T.O.
2. Immunologic mechanisms in intestinal disease;Targan, SR;Ann Intern Med,1987
3. Examination of the low proliferative capacity of human jejunal intraepithelial lymphocytes. Clin Exp Immunol;Ebert, E.C.; Roberts, A.I.; Brolin, R.E.; Raska, K.,1986
4. Proliferative response of human intraepithelial lymphocytes to various T cell stimuli;Ebert, E.C.;Gastroenterology,1989
5. Human intestinal mononuclear cells. I. Investigation of antibody-dependent, lectin-induced this cell popullation. The slow reaction of TEL after triggering could be the result of impairment by the isolation procedure, but peripheral blood lymphocytes treated in the same way do not show delayed toxic function. Another reason may be the mechanism of cytotoxicity. The mechanism and spontaneous cell-mediated cytotoxic capabilities;MacDermott, R.P.; Franklin, G.O.; Jenkins, K.M.; Kodner, I.J.; Nash, G.S.; Weintrieb, I.J.;Gastroenterology,1980