1. BROOKSBY, I. A. B., JENKINS, B. S., COLTART, D. J., WEBB-PEPLOE, M. M., and DAVIES, M. J. (1974) Lancet, 2, 1222
2. CAMPBELL, M., and SHACKLE, J. W. (1932) Brit. med.J_., 1, HEGGTVEIT, H. A. (1964) Circulation, 29, 346
3. Brit. J_. vener;INCE, W.E.; MAHABIR, B.S.;Dis,1974
4. Appendix Case reports;SAMAAN, H.A.;Surg. (Torino),1973
5. Case 2, a 57-year-old white female presented in January 1972 with a 6-month history of paroxysmal dyspnoea, effort dyspnoea, and angina. Clinically there was severe aortic regurgitation. The VDRL, RPCF, FTA-ABS, treponemal immobilization (TPI) tests were all negative, but a chest radiograph showed cardiac enlargement with a dilated calcified ascending aorta and the electrocardiogram indicated left axis deviation and left ventricular hypertrophy with strain. Aortography showed severe aortic regurgitation with aneurysmal dilatation of the ascending aorta. At operation (24.3.72) the aneurysm of the ascending aorta and the dilated aortic root were confirmed. The coronary ostia were normal. The valve and ascending aorta were replaced using a No. 10 StarrEdwards prosthesis and a 35 mm Dacron graft. The postoperative course was uneventful and the patient has remained well with minimal effort dyspnoea for the last 3 years. Histology of the aorta confirmed the presence of syphilis and the aortic cusps were normal