1. affecting the treatment group in the 24 hours immediately following treatment. This difference is no longer apparent in the subsequent 24 hour period. In 1988, Ment reported no clinically important renal abnormalities but provides no data.3' Bada33 provided data that show significant changes in plasma creatinine and sodium concentrations, osmolalities, and urine output, but comments that these differences were not abnormal - that is, not clinically important
2. Increased risk of bronchopulmonary dysplasia in infants with patent ductus arteriosus;Brown, E.R.;J Pediatr,1979
3. Patent ductus arteriosus in premature infants: incidence and relation to pulmonary disease and management;Kitterman, J.A.; LH, Jr., Edmunds; Gregory, G.A.; Heymann, M.A.; Tooley, W.H.; Rudolph, A.M.;N Eng J Med,1972
4. Closure of the ductus arteriosus in premature infants by inhibition of prostaglandin synthesis;Heymann, M.A.; Rudolph, A.M.; Silverman, N.H.;NEnglJMed,1976
5. Abnormal cerebral haemodynamics in preterm infants with patent ductus arteriosus;Lipman, B.; Server, G.A.; Brazy, J.E.;Pediatrics,1982