1. once rickets is diagnosed treatment is usually given. An impaired conversion of vitamin D to 25-hydroxy vitamin D might be responsible for the development of rickets in neonatal hepatitis to a greater extent than an impaired absorption of vitamin D and bone building materials. The suggestion has been made that 'preterm delivery may also have contributed to the development of rickets' in 2 infants with neonatal jaundice (Thomas and Glasgow, 1974);all;these substances are stored in the body, the occurrence of manifest bone disease might very well depend on the size of the infant's stores. In the few reports where gestational age at delivery is given, actually all the infants with neonatal jaundice and rickets were premature, in contrast with the full maturity of the infants with the combination of rickets and biliary atresia (Yu; et al,1971
2. Serum alkaline phosphatase in infants with obstructive jaundice; relation to vitamin D supplementation;Bastis-Maounis, B.; Matsaniotis, N.; Maounis, F.;Journal of Pediatrics,1973
3. 25hydroxycholecalciferol dynamics in human plasma. Abstract No. 597, IV International Congress of Endocrinology. Excerpta Medica;Bayard, J.; Bec, P.; Louvet, J.P.; Boulard, C.,1972
4. Dystrophie osteomalacique du pr6matur6;Boissiere, H.; Cagnat, R.; Poissonier, M.; d'Angely, S.;Annales de Pediatrie,1964
5. Bone disease in infants and children with hepatobiliary disease;Kobayashi, A.; Kawai, S.; Utsunomiya, T.; Ohbe, Y.;Archives of Disease in Childhood,1974