1. an overall reduction in myocardial oxygen consump-Nifedipine therapy for stable angina pectoris: prelimition, presumably caused by its peripheral nary results of effects on angina frequency and treadmill haemodynamic actions. This in turn leads to a exercise response;Am J Cardiol,1979
2. Clinical experiences with a new coronary-active substance (BAYI1040). Arneim Forsch;Kobayashi, T.; Ito, Y.; Tawara, I.,1972
3. Effect of nifedipine on exercise tolerance in patients with angina pectoris;Ekelund, L.G.; Atterhog, J.H.; Melin, A.L.,1975
4. oxygen consumption, coronary sinus flow, or coro-4 Antman;E, Muller J.; S, Goldberg;N EnglJ3 Med; Nifedipine for nary vascular resistance. It did, however, produce a significant fall in coronary arteriovenous oxygen difference, both in sinus rhythm and during atrial pacing, which points towards a direct coronary vasodilating action. Direct comparisons of the effects of the two drugs showed a significant difference in coronary sinus flow and coronary vascular resistance in sinus coronary artery spasm. Experience in 127 patients,1980
5. 5-dicarbonsauredimethylester Nifedipine) Bay a 1040;Vater, W.; Kroneberg, G.; Hoffmeister, F.;Zur Pharmokologie von,1972