Serum increase and liver overexpression of carbohydrate 19.9 antigen in patients with genetic haemochromatosis.

Author:

Deugnier Y M,Rabot A F,Guyader D,Moirand R,Turlin B,Boucher E,Lebert P,Brissot P

Publisher

BMJ

Subject

Gastroenterology

Reference39 articles.

1. Characterization of CA19.9 bearing mucins as physiological exocrine pancreatic secretion products. haemochromatosis cirrhosis may suggest such an impairment. In addition, circulating serum Cancer;Kalthoff, H.; Kreiker, C.; Schmiegel, W.H.; Greten, H.; Thiele, H.G.;Res,1986

2. Composition and production of pancreatic tumor related antigens;Appert, H.E.;Int Jf Pancreatol,1990

3. The carbohydrate antigenic and chronic liver diseases even in the absence of cirrhosis.38-40 Therefore, it can be suggested determinant 19.9 (CAl9. 9): a monoclonal antibody defined tumor marker;BC, Del Villano; VR, Zurawski,1983

4. Biosynthesis of the cancer-associated sialyl-le(a) antigen;Hansson, G.C.; Zopf, D.;J Biol Chem,1985

5. A monoclonal antibody-defined antigen associated with gastrointestinal cancer is a ganglioside containing sialylated lactoN-fucopentaoseIl;Magnani, J.L.; Nilsson, B.; Brockhaus, M.;JBiol Chem; catabolism. Besides a putative role of iron in itself, a role of iron related liver damage may be discussed. Such a distinction is difficult to make, however, as the increase in serum CA 19.9 was correlated both with the amount of iron excess and,1982

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