Pseudohyperaldosteronism induced by habitual ingestion of liquorice.

Author:

Holmes A. M.,Young J.,Marrott P. K.,Prentice E.

Publisher

BMJ

Subject

General Medicine

Reference29 articles.

1. Metabolic studies, aldosterone secretion rate and plasma renin after carbenoxolone sodium;BARON, J.H.; NABARRO, J.D.N.; SLATER, J.D.H.; TUFFLEY, R.;British Medical Journal,1969

2. De uitscheiding van water en electrolyten gedurende het elmaal in onder invloed van succus liquiritiae;BORST, J.C.G.; BLONHERT, G.; MOLHUYSEN, J.A.; GERBRANDY, J.; TURNER, K.; VRIES, D.E.; A, L.;Acta clinica Belgica,1950

3. Estimation of plasma renin;BROWN, J.J.; DAVIES, D.L.; LEVER, A.F.; ROBERTSON, J.I.S.; TREE, M.;Biochemical Journal,1964

4. The cause of hypokalaemia (hypopotassaemia following para-aminosalicylate therapy);CAMPBELL, A.H.; NEUFELD, O.E.;Medical Journal of Australia,1951

5. Potassium deficiency in p-aminosalicylic acid therarpy;CAYLEY, F.E.D.E.W.;Lancet,1950

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2. Glycyrrhiza glabra;Edible Medicinal and Non-Medicinal Plants;2016

3. The cardiovascular complications of licorice;Cardiovascular Endocrinology;2013-09

4. Licorice abuse: time to send a warning message;Therapeutic Advances in Endocrinology and Metabolism;2012-07-12

5. TOXIC MYOPATHIES;Neurologic Clinics;1997-08

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