Atrophic gastric changes in both Helicobacter felis and Helicobacter pylori infected mice are host dependent and separate from antral gastritis.

Author:

Sakagami T,Dixon M,O'Rourke J,Howlett R,Alderuccio F,Vella J,Shimoyama T,Lee A

Publisher

BMJ

Subject

Gastroenterology

Reference23 articles.

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2. Molecular characterization of the 128-kDa immunodominant antigen of Helicobacter pylon associated with cytotoxicity;Covacci, A.; Censini, S.; Bugnoli, M.; Petracca, R.; Burroni, D.; Macchia, G.,1993

3. Cloning and expression of a high-molecular-mass major antigen of Helicobacter pylori - evidence of linkage to cytotoxin production;Tummuru, M.K.R.; Cover, T.L.; Blaser, M.J.;Infect Immun,1993

4. Helicobacter pylon and gastric mucosal cytokines - evidence that cagA-positive strains are more virulent;Crabtree, J.E.; Farmery, S.M.;Lab Invest,1995

5. Helicobacter pylon induced interleukin-8 expression in gastric epithelial cells is associated with CagA positive phenotype;CrabtreeJE, CovacciA; FarmerySM, XiangZ; TompkinsDS, Perry S.;J Clin Pathol,1995

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