1. Breakdown of the blood-brain barrier precedes symptoms and other MRI signs of new lesions in multiple sclerosis. Brain nificantly raised CRP concentrations in patients with inactive MRI compared with;Kermode, A.G.; Thompson, A.J.; Tofts, P.,1990
2. Duration and selectivity of blood-brain barrier breakdown in controls that could not be explained by infection, implying the presence of active inflamchronic relapsing experimental allergic encephalomyelitis studied by gadolinium-DTPA and protein markers;Hawkins, C.P.; Munro, P.M.G.; Mackenzie, F.;Brain,1990
3. Correlation between magnetic resonance imaging findings and lesion development in chronic, active multiple sclerosis. Ann of the MRI protocol used in this study. This finding supports a pathological study in which;Katz, D.; Taubenberger, J.K.; Cannella, B.;Neurol,1993
4. Oligodendrocyte injury is an early event in lesions of multhe number of histologically active lesions far exceeded the number detected by postmortem tiple sclerosis;Rodriguez, M.; Scheithauer, B.W.; Forbes, G.; Kelly, P.J.,1993
5. Immunohistochemical analysis of the cellular infiltrate in multiple sclerosis T2 weighted MRI.44 C Reactive protein is synthesised by the liver mainly in response to IL6, lesions;Hauser, S.L.; Bhan, A.K.; Gilles, F.;Ann Neurol,1986