Absence of antibodies stimulating H2-receptor mediated cyclic adenosine monophosphate (cAMP) production in peptic ulcer disease.

Author:

Sarem-Aslani A,Bergmann C,Walker S,Ratge D,Klotz U,Wisser H

Publisher

BMJ

Subject

Gastroenterology

Reference9 articles.

1. Gastric cell c-AMP stimulating autoantibodies in duodenal ulcer disease;F, De Lazzari; R, Mirakian; L, Hammond; C, Venturi; R, Naccarato; GF, Bottazzo;Gut,1988

2. Characterization of a newly established human gastric cancer cell line HGT-1 bearing histamine H, receptors;Laboisse, C.L.; Augeron, C.; Coutourier-Turpin, M.-H.; Gespach, C.; Potet, F.; Cheret, A.-M.;Cancer Res,1982

3. Histamine and VIP interactions with receptor-cyclic AMP systems in the human gastric cancer cell line HGT-1;Emami, S.; Gespach, C.; Forgue-Lafitte, M.-E.; Broer, Y.; Rosselin, G.;Life Sci,1983

4. Effect of truncated glucagon-like peptide 1 on cAMP in rat gastric glands and HGT-1 human gastric cancer cells;Hansen, A.B.; Gespach, C.P.; Rosselin, G.E.; Holst, J.J.;FEBS Lett,1988

5. Frequent non-response to H,-receptor antagonists in cirrhotics;Walker, S.; Krishna, D.R.; Klotz, U.; Bode, J.C.;Gut,1989

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