In vivo binding of circulating immune complexes by C3b receptors (CR1) of transfused erythrocytes.

Author:

Inada Y,Kamiyama M,Kanemitsu T,Ikegami H,Watanabe K,Clark W S,Asai Y

Publisher

BMJ

Subject

General Biochemistry, Genetics and Molecular Biology,Immunology,Immunology and Allergy,Rheumatology

Reference25 articles.

1. Kinetics of interaction of immune complexes with complement receptors on human red blood cells: modification of complexes during interaction with red cells. Clin Exp Immunol;E, Medof M.; M, Prince G.; J-F, Oger J.,1982

2. Ability of complement to release systemic lupus erythematosus immune complexes from cell receptors;E, Medof M.; D, Scarborough; G, Miller;Clin Exp Immunol,1981

3. Complement receptor is an inhibitor of complement cascade;lida K; V, Nussenzweig;J Exp Med,1981

4. Identification of the membrane glycoprotein that is the C3b receptor of the human erythrocyte, polymorphonuclear leukocyte, B lymphocyte and monocyte;T, Fearon D.;J Exp Med,1980

5. Defective immune-adherence (C3b) receptor on erythrocytes from patients with systemic lupus erythematosus;Miyakawa, Y.; Yamada, A.; Kasaka, K.; Tsuda, F.; Kosugi, E.; Mayumi, M.;Lancet,1981

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