Dysregulated KLF4 expression plays a pivotal role in the pathogenesis of pancreatic intraductal papillary mucinous neoplasms-Reference-Cited by-同舟云学术

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Dysregulated KLF4 expression plays a pivotal role in the pathogenesis of pancreatic intraductal papillary mucinous neoplasms

Published:2024-07-05 Issue: Volume: Page:gutjnl-2024-332255
ISSN:0017-5749
Container-title:Gut
language:en
Short-container-title:Gut

Author:

Zuo Xiangsheng,Wang Liang,Liu Yi,Wang Huamin,Hafley Margarete,Gagea Mihai,Chen Ru,Xiong Yun,Pan Sheng,Shureiqi Imad,Bresalier Robert S,Wei Daoyan^ORCID

Funder

National Institutes of Health, NCI.

Publisher

BMJ

Reference10 articles.

1. Multiregion whole-exome sequencing of intraductal papillary mucinous neoplasms reveals frequent somatic KLF4 mutations predominantly in low-grade regions

2. Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential

3. KLF4 Is Essential for Induction of Cellular Identity Change and Acinar-to-Ductal Reprogramming during Early Pancreatic Carcinogenesis

4. Sox9+ ductal cells are multipotent progenitors throughout development but do not produce new endocrine cells in the normal or injured adult pancreas

5. SOX9: a useful marker for pancreatic ductal lineage of pancreatic neoplasms

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