Transforming growth factor beta in normal human lung: preferential location in bronchial epithelial cells.

Author:

Magnan A,Frachon I,Rain B,Peuchmaur M,Monti G,Lenot B,Fattal M,Simonneau G,Galanaud P,Emilie D

Publisher

BMJ

Subject

Pulmonary and Respiratory Medicine

Reference28 articles.

1. Targeted disruption of the mouse transforming growth factor-,1 gene results in multifocal inflammatory disease;Shull, M.M.; Ormsby, I.; Kier, A.B.; Pawlowski, S.; Diebold, R.J.; Yin, M.;Nature,1992

2. null mutation in mice causes excessive inflammatory;Kulkarni, A.B.; Huh, C.G.; Becker, D.; Geiser, A.; Lyght, M.; Flanders, K.C.,1993

3. High levels of transforming growth factor-f are present in the epithelial lining fluid of the normal human lower respiratory tract;Yamauchi, K.; Martinet, Y.; Basset, P.; Fells, G.A.; Crystal, R.G.;Am Rev Respir Dis,1988

4. Expression and secretion of transforming growth factor-f by activated human macrophages;Assoian, R.K.; Fleurdelys, B.E.; Stevenson, H.C.; Miller, P.J.; Madtes, D.K.; Raines, E.W.;Proc Natl Acad Sci USA,1987

5. Polymerase chain reaction amplification of messages for growth factors in cells from human bronchoalveolar lavage fluids;Adolff, C.H.; Golden, J.A.; Kennedy, P.W.; Goetzl, E.J.; Turck, C.W.;Inflammation,1991

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