Response to: Correspondence on ‘Mechanical overloading promotes chondrocyte senescence and osteoarthritis development through downregulating FBXW7’ by Loeser et al
Author:
Funder
National Natural Science Foundation of China
Natural Science Foundation of Guangdong Province
Publisher
BMJ
Subject
General Biochemistry, Genetics and Molecular Biology,Immunology,Immunology and Allergy,Rheumatology
Reference29 articles.
1. Mechanical overloading promotes chondrocyte senescence and osteoarthritis development through downregulating FBXW7
2. Activation of stress-activated protein kinase in osteoarthritic cartilage: evidence for nitric oxide dependence
3. C-Jun transactivates Puma gene expression to promote osteoarthritis;Lu;Mol Med Rep,2014
4. SP600125, a JNK-Specific inhibitor, regulates in vitro auricular cartilage regeneration by promoting cell proliferation and inhibiting extracellular matrix metabolism;Zhang;Front Cell Dev Biol,2021
5. Adiponectin is a potential catabolic mediator in osteoarthritis cartilage
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