Imputation-based analysis of MICA alleles in the susceptibility to ankylosing spondylitis
Author:
Publisher
BMJ
Subject
General Biochemistry, Genetics and Molecular Biology,Immunology,Immunology and Allergy,Rheumatology
Reference8 articles.
1. Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility
2. Progress of genome-wide association studies of ankylosing spondylitis;Li;Clin Transl Immunology,2017
3. Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci
4. Major histocompatibility complex associations of ankylosing spondylitis are complex and involve further epistasis with ERAP1
5. Associations of MICA Polymorphisms with Inflammatory Rheumatic Diseases;Wang;Open Rheumatol J,2015
Cited by 15 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Exploring the Pathogenesis of Spondylarthritis beyond HLA-B27: A Descriptive Review;International Journal of Molecular Sciences;2024-05-31
2. High population frequencies of MICA copy number variations originate from independent recombination events;Frontiers in Immunology;2023-11-15
3. Genotype by sex interactions in ankylosing spondylitis;Nature Genetics;2023-01
4. Reply to: Genotype by sex interactions in ankylosing spondylitis;Nature Genetics;2023-01
5. HLA-B27 and not variation in MICA is responsible for genotype by sex interaction in risk of Ankylosing Spondylitis;2021-12-17
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