1. The first hour ofacute myocardial infarction observed dunng mobile intensive care;Adgey, A.A.J.; Webb, S.W.,1985
2. Neurogenic control ofthe heart or sympathble mechanism for reduced variation in RR interval etic-vagal interaction. In: Riemersma RA, Oliver during anterior infarction is the higher density of parasympathetic innervation to the posterior and inferior left ventricular walls2' and known preponderance of parasympathetically mediated reflex activity during ischaemia or infarction of these regions.2' Such dependence of autonomic responses upon site of infarction was shown experimentally after acute coronary occlusions in the cat or dog.21 22 MF, eds. Catecholanines in the non ischaemic and ischaemic myocardiwn;Kent, K.M.,1982
3. Relationship between sympathetic neural activity, coronary dynamics and vulnerability to ventricular fibrillation during myocardial ischaemia and reperfusion;Lombardi, F.; Verrier, R.L.; Lown, B.;Am Heart J,1983
4. Practical bedside investigation of diabetic Differential responses atrributable to the preferential location of vagal afferents in the inferior and posautonomic failure;Ewing, D.J.,1983
5. hour electrcardiograms. Br Heart J 1984;S2: 396-402.