Author:
Benson M D,Turpin J C,Lucotte G,Zeldenrust S,LeChevalier B,Benson M D
Subject
Genetics (clinical),Genetics
Reference11 articles.
1. In: The metabolic basis of inherited disease;Benson, M.D.; Amyloidosis, Wallace M.R.,1989
2. A point mutation in transthyretin increases affinity for thyroxine and produces euthyroid hyperthyroxinemia;Moses, A.C.; Rosen, H.N.; Moller, D.E.;7 Clin Invest,1990
3. An inherited nonamyloidogenic transthyretin variant, [Ser61-TTR, with increased thyroxine-binding affinity, characterized by DNA sequencing;Fitch, N.J.S.; Akbari, M.T.; Ramsden, D.B.;J Endocrinol,1991
4. Prealbumin and retinol binding protein serum concentrations in the Indiana type hereditary amyloidosis;Benson, M.D.; Dwulet, F.E.;Arthritis Rheum,1983
5. Comparison of two methods of high molecular-weight DNA isolation from human leucocytes;Gautreau, C.; Rahuel, C.; Cartron, J.P.; Lucotte, G.;Anal Biochem,1983
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