Abnormalities in exercising skeletal muscle in congestive heart failure can be explained in terms of decreased mitochondrial ATP synthesis, reduced metabolic efficiency, and increased glycogenolysis.

Author:

Kemp G. J.,Thompson C. H.,Stratton J. R.,Brunotte F.,Conway M.,Adamopoulos S.,Arnolda L.,Radda G. K.,Rajagopalan B.

Publisher

BMJ

Subject

Cardiology and Cardiovascular Medicine

Reference35 articles.

1. Skeletal muscle metabolism in heart failure: a 31P nuclear magnetic resonance spectroscopy study of leg muscle;Arnolda, L.; Conway, M.; Dolecki, M.; Sharif, H.; Rajagopalan, B.; Ledingham, J.G.G.;Clin Sci,1990

2. Contribution of skeletal muscle atrophy to exercise intolerance and altered muscle metabolism in heart failure;Mancini, D.M.; Walter, G.; Reichek, N.; Lenkinski, R.; McCully, K.K.; Mullen, J.L.;Circulation; Skeletal muscle bioenergetics in heart failure,1992

3. Contribution of specific skeletal muscle metabolic abnormalities to limitation of exercise capacity in patients with chronic heart failure: a phosphorus 31 nuclear magnetic resonance;Chati, Z.; Zannad, F.; Robin-Lherbier, B.; Escanye, J.M.; Jeandel, C.; Robert, J.;Am Heart Jf,1994

4. Skeletal muscle metabolism in patients with congestive heart failure: relation to clinical severity and blood flow;Massie, B.; Conway, M.; Yonge, R.; Frostick, S.; Sleight, P.; Ledingham, J.G.G.;Circulation,1987

5. Alterations of skeletal muscle metabolism in human studied by phosphorus 31 magnetic resonance spectroscopy in congestive heart failure;Rajagopalan, B.; Conway, M.A.; Massie, B.; Radda, G.K.;Am Jf Cardiol,1988

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