Behaviour of acid secretion, gastrin release, serum pepsinogen I, and gastric emptying of liquids over six months from eradication of helicobacter pylori in duodenal ulcer patients. A controlled study.

Author:

Parente F,Maconi G,Sangaletti O,Minguzzi M,Vago L,Bianchi Porro G

Publisher

BMJ

Subject

Gastroenterology

Reference33 articles.

1. Helicobacter pylori infection in peptic ulcer disease;Berstad, K.; Berstad, A.,1993

2. Campylobacter pylori and duodenal ulcer; the gastrin link;Levi, S.; Beardshall, K.; Haddad, G.; Playford, R.; Ghosh, P.; Calam, J.;Lancet,1989

3. Inappropriate hypergastrinaemia in asymptomatic healthy subjects infected with Helicobacter pylori;Smith, J.T.L.; Pounder, R.E.; Nwokolo, C.U.; Lanzon-Miller, S.; Evans, D.G.; Graham, D.Y.,1990

4. Ablation of exaggerated meal stimulated gastrin release in duodenal ulcer patients after clearance of Helicobacter (Campylobacter) pylori infection;Graham, D.Y.; Opekun, A.; Lew, G.M.; DJ, Jr, Evans; Klein, P.D.; Evans, D.G.;Am J Gastroenterol,1990

5. The degree of hypergastrinaemia induced by Helicobacter pylori is the same in duodenal ulcer patients and asymptomatic volunteers;Chittajallu, R.S.; Ardill, J.E.S.; McColl, K.E.L.

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