Genetic basis of glioma progression
Author:
Affiliation:
1. International Agency for Research on Cancer (IARC)
Publisher
Japan Academy
Subject
General Physics and Astronomy,General Agricultural and Biological Sciences,General Medicine
Link
https://www.jstage.jst.go.jp/article/pjab/79B/3/79B_3_78/_pdf
Reference76 articles.
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2. 2) Biernat, W., Kleihues, P., Yonekawa, Y., and Ohgaki, H. (1997) Amplification and overexpression of MDM2 in primary (de novo) glioblastomas. J. Neuropathol. Exp. Neurol. 56, 180-185.
3. 3) Biernat, W., Tohma, Y., Yonekawa, Y., Kleihues, P., and Ohgaki, H. (1997) Alterations of cell cycle regulatory genes in primary (de novo) and secondary glioblastomas. Acta Neuropathol. 94, 303-309.
4. 4) Tohma, Y., Gratas, C., Biernat, W., Peraud, A., Fukuda, M., Yonekawa, Y., Kleihues, P., and Ohgaki, H. (1998) PTEN (MMAC1) mutations are frequent in primary glioblastomas (de novo) but not in secondary glioblastomas. J. Neuropathol. Exp. Neurol. 57, 684-689.
5. 5) Fujisawa, H., Reis, R. M., Nakamura, M., Colella, S., Yonekawa, Y., Kleihues, P., and Ohgaki, H. (2000) Loss of heterozygosity on chromosome 10 is more extensive in primary (de novo) than in secondary glioblastomas. Lab. Invest. 80, 65-72.
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1. Population-Based Studies on Incidence, Survival Rates, and Genetic Alterations in Astrocytic and Oligodendroglial Gliomas;Journal of Neuropathology & Experimental Neurology;2005-06
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