GD3-and O-acetylated GD3-gangliosides in the GM2 synthase-deficient mouse brain and their immunohistochemical localization
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Published:2006
Issue:6
Volume:82
Page:189-196
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ISSN:0386-2208
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Container-title:Proceedings of the Japan Academy, Series B
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language:en
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Short-container-title:Proceedings of the Japan Academy. Ser. B: Physical and Biological Sciences
Author:
MATSUDA Junko1, VANIER Marie T.2, POPA Iuliana3, PORTOUKALIAN Jacques3, SUZUKI Kunihiko1
Affiliation:
1. Institute of Glycotechnology, Future Science and Technology Joint Research Center, Tokai University 2. INSERM U 499, RTH Laënnec School of Medicine, and Fondation Gillet-Mérieux, Lyon-Sud Hospital 3. EA-3732 University of Lyon-1, Edouard Herriot Hospital
Subject
General Physics and Astronomy,General Agricultural and Biological Sciences,General Medicine
Reference46 articles.
1. 1) Nagata, Y., Yamashiro, S., Yodoi, J., Lloyd, K. O., Shiku, H., and Furukawa, K. (1992) Expression cloning of β1,4 N-acetylgalactosaminyltransferase cDNAs that determine the expression of GM2 and GD2 gangliosides. J. Biol. Chem. 267, 12082-12089. 2. 2) Sango, K., Johnson, O. N., Kozak, C. A., and Proia, R. L. (1995) β-1,4-N-acetylgalactosaminyltransferase involved in ganglioside synthesis: cDNA sequence, expression, and chromosome mapping of the mouse gene. Genomics 27, 362-365. 3. 3) Takamiya, K., Yamamoto, A., Furukawa, K., Yamashiro, S., Shin, M., Okada, M., Fukumoto, S., Haraguchi, M., Takeda, N., Fujimura, K. et al. (1996) Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system. Proc. Natl. Acad. Sci. USA 93, 10662-10667. 4. 4) Liu, Y. J., Wada, R., Kawai, H., Sango, K., Deng, C. X., Tai, T., McDonald, M. P., Araujo, K., Crawley, J. N., Bierfreund, U. et al. (1999) A genetic model of substrate deprivation therapy for a glycosphingolipid storage disorder. J. Clin. Invest. 103, 497-505. 5. 5) Sheikh, K. A., Sun, J., Liu, Y. J., Kawai, H., Crawford, T. O., Proia, R. L., Griffin, J. W., and Schnaar, R. L. (1999) Mice lacking complex gangliosides develop Wallerian degeneration and myelination defects. Proc. Natl. Acad. Sci. USA 96, 7532-7537.
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