The effect of folic acid supplementation on damage caused by chronic cerebral hypo-perfusion in the hippocampus: the role of antioxidant agents and homocysteine
Author:
Publisher
Springer Science and Business Media LLC
Subject
Public Health, Environmental and Occupational Health,Nutrition and Dietetics,Physiology,Biochemistry,Endocrinology, Diabetes and Metabolism,Food Science
Link
https://link.springer.com/content/pdf/10.1186/s41110-023-00240-9.pdf
Reference26 articles.
1. Zhao Y, Gong CX. From chronic cerebral hypoperfusion to Alzheimer-like brain pathology and neurodegeneration. Cell Mol Neurobiol. 2015;35(1):101–10. https://doi.org/10.1007/s10571-014-0127-9.
2. Yin G, Gan Y, Jiang H, Yu T, Liu M, Zhang Y, et al. Direct quantification and visualization of homocysteine, cysteine, and glutathione in Alzheimer’s and Parkinson’s disease model tissues. Anal Chem. 2021;93(28):9878–86. https://doi.org/10.1021/acs.analchem.1c01945.
3. Ciacciarelli A, Sette G, Giubilei F, Orzi F. Chronic cerebral hypoperfusion: an undefined, relevant entity. J Clin Neurosci. 2020;73:8–12. https://doi.org/10.1016/j.jocn.2020.01.026.
4. Daulatzai MA. Cerebral hypoperfusion and glucose hypometabolism: key pathophysiological modulators promote neurodegeneration, cognitive impairment, and Alzheimer’s disease. J Neurosci Res. 2017;95(4):943–72. https://doi.org/10.1002/jnr.23777.
5. Miranpuri GS, Nguyen J, Moreno N, Yutuc N, Kim J, Butter S, et al. Folic acid modulates matrix metalloproteinase-9 expression following spinal cord injury. Ann Neurosci. 2019;26(2):60–5. https://doi.org/10.5214/ans.0972.7531.260205.
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