Structural biology of presenilin 1 complexes-Reference-Cited by-同舟云学术

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Structural biology of presenilin 1 complexes

Published:2014-12 Issue:1 Volume:9 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Li Yi,Bohm Christopher,Dodd Roger,Chen Fusheng,Qamar Seema,Schmitt-Ulms Gerold,Fraser Paul E,St George-Hyslop Peter H

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Molecular Biology

Link

https://link.springer.com/content/pdf/10.1186/1750-1326-9-59.pdf

Reference88 articles.

1. De Strooper B, Iwatsubo T, Wolfe MS: Presenilins and gamma-secretase: structure, function, and role in Alzheimer disease. Cold Spring Harb Perspect Med. 2012, 2: a006304-

2. Li H, Wolfe MS, Selkoe DJ: Toward structural elucidation of the gamma-secretase complex. Structure. 2009, 17: 326-334.

3. Haass C, Kaether C, Thinakaran G, Sisodia S: Trafficking and proteolytic processing of APP. Cold Spring Harb Perspect Med. 2012, 2: a006270-

4. Wolfe MS: Processive proteolysis by gamma-secretase and the mechanism of Alzheimer's disease. Biol Chem. 2012, 393: 899-905.

5. Chow VW, Mattson MP, Wong PC, Gleichmann M: An overview of APP processing enzymes and products. Neuromolecular Med. 2010, 12: 1-12.

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1. Gamma Secretase as an Important Drug Target for Management of Alzheimer’s Disease: A Comprehensive Review;Current Topics in Medicinal Chemistry;2024-01

2. Probing the Mechanisms of Inhibitors Binding to Presenilin Homologue Using Molecular Dynamics Simulations;Molecules;2023-02-22

3. Associations of Naturally Occurring Antibodies to Presenilin-1 with Brain Amyloid-β Load and Cognitive Impairment in Alzheimer’s Disease;Journal of Alzheimer's Disease;2022-12-06

4. ER stress and UPR in Alzheimer’s disease: mechanisms, pathogenesis, treatments;Cell Death & Disease;2022-08-15

5. The novel I213S mutation in PSEN1 gene is located in a hotspot codon associated with familial early-onset Alzheimer's disease;Neurobiology of Aging;2022-04

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