Integrative proteomic analysis of the NMDA NR1 knockdown mouse model reveals effects on central and peripheral pathways associated with schizophrenia and autism spectrum disorders-Reference-Cited by-同舟云学术

Integrative proteomic analysis of the NMDA NR1 knockdown mouse model reveals effects on central and peripheral pathways associated with schizophrenia and autism spectrum disorders

Published:2014-07-04 Issue:1 Volume:5 Page:
ISSN:2040-2392
Container-title:Molecular Autism
language:en
Short-container-title:Molecular Autism

Author:

Wesseling Hendrik,Guest Paul C,Lee Chi-Ming,Wong Erik HF,Rahmoune Hassan,Bahn Sabine

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Developmental Biology,Developmental Neuroscience,Molecular Biology

Link

https://link.springer.com/content/pdf/10.1186/2040-2392-5-38.pdf

Reference118 articles.

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3. Eastwood SL, Kerwin RW, Harrison PJ: Immunoautoradiographic evidence for a loss of alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate-preferring non-N-methyl-D-aspartate glutamate receptors within the medial temporal lobe in schizophrenia. Biol Psychiatry. 1997, 41: 636-643.

4. Tamminga CA: Schizophrenia and glutamatergic transmission. Crit Rev Neurobiol. 1998, 12: 21-36.

5. Lahti AC, Koffel B, LaPorte D, Tamminga CA: Subanesthetic doses of ketamine stimulate psychosis in schizophrenia. Neuropsychopharmacology. 1995, 13: 9-19.

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