Absence of Appl2 sensitizes endotoxin shock through activation of PI3K/Akt pathway-Reference-Cited by-同舟云学术

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Absence of Appl2 sensitizes endotoxin shock through activation of PI3K/Akt pathway

Published:2014-10-02 Issue:1 Volume:4 Page:
ISSN:2045-3701
Container-title:Cell & Bioscience
language:en
Short-container-title:Cell Biosci

Author:

Mao Liufeng,Lin Wanhua,Nie Tao,Hui Xiaoyan,Gao Xuefei,Li Kuai,Ding Mengxiao,Tang Xiaofeng,Li Peng,Wang Yu,Xu Aimin,Liu Pentao,Wu Donghai

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology

Link

https://link.springer.com/content/pdf/10.1186/2045-3701-4-60.pdf

Reference39 articles.

1. Martin GS, Mannino DM, Eaton S, Moss M: The epidemiology of sepsis in the United States from 1979 through 2000. N Engl J Med. 2003, 348: 1546-1554. 10.1056/NEJMoa022139

2. Ulevitch RJ, Tobias PS: Receptor-dependent mechanisms of cell stimulation by bacterial endotoxin. Annu Rev Immunol. 1995, 13: 437-457. 10.1146/annurev.iy.13.040195.002253

3. May MJ, Ghosh S: Signal transduction through NF-kappa B. Immunol Today. 1998, 19: 80-88. 10.1016/S0167-5699(97)01197-3

4. Brown K, Gerstberger S, Carlson L, Franzoso G, Siebenlist U: Control of I kappa B-alpha proteolysis by site-specific, signal-induced phosphorylation. Science. 1995, 267: 1485-1488. 10.1126/science.7878466

5. Chen Z, Hagler J, Palombella VJ, Melandri F, Scherer D, Ballard D, Maniatis T: Signal-induced site-specific phosphorylation targets I kappa B alpha to the ubiquitin-proteasome pathway. Genes Dev. 1995, 9: 1586-1597. 10.1101/gad.9.13.1586

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