CD36 overexpression: a possible etiopathogenic mechanism of atherosclerosis in patients with prediabetes and diabetes-Reference-Cited by-同舟云学术

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CD36 overexpression: a possible etiopathogenic mechanism of atherosclerosis in patients with prediabetes and diabetes

Published:2017-07-18 Issue:1 Volume:9 Page:
ISSN:1758-5996
Container-title:Diabetology & Metabolic Syndrome
language:en
Short-container-title:Diabetol Metab Syndr

Author:

Lopez-Carmona M. D.,Plaza-Seron M. C.,Vargas-Candela A.,Tinahones F. J.,Gomez-Huelgas R.,Bernal-Lopez M. R.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/content/pdf/10.1186/s13098-017-0253-x.pdf

Reference48 articles.

1. Cooper R, Cutler J, Desvigne-Nickens P, Fortmann SP, Friedman L, Havlik R, et al. Trends and disparities in coronary heart disease, stroke, and other cardiovascular diseases in the United States: findings of the national conference on cardiovascular disease prevention. Circulation. 2000;102(25):3137–47.

2. Ford ES. Trends in predicted 10-year risk of coronary heart disease and cardiovascular disease among US adults from 1999 to 2010. J Am Coll Cardiol. 2013;61(22):2249–52.

3. Executive summary. Standards of medical care in diabetes–2010. Diabetes Care. 2010;33(Suppl1):S4–10.

4. Park YM, Kashyap SR, Major JA, Silverstein RL. Insulin promotes macrophage foam cell formation: potential implications in diabetes-related atherosclerosis. Lab Invest. 2012;92(8):1171–80.

5. The DECODE study group, European Diabetes Epidemiology Group. Glucose tolerance and mortality: comparison of WHO and American Diabetes Association diagnostic criteria. Diabetes epidemiology: Collaborative analysis Of Diagnostic criteria in Europe. Lancet. 1999;354(9179):617–21.

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