Exenatide induces aortic vasodilation increasing hydrogen sulphide, carbon monoxide and nitric oxide production-Reference-Cited by-同舟云学术

Exenatide induces aortic vasodilation increasing hydrogen sulphide, carbon monoxide and nitric oxide production

Published:2014-04-02 Issue:1 Volume:13 Page:
ISSN:1475-2840
Container-title:Cardiovascular Diabetology
language:en
Short-container-title:Cardiovasc Diabetol

Author:

Sélley Eszter,Kun Szilárd,Szijártó István András,Laczy Boglárka,Kovács Tibor,Fülöp Ferenc,Wittmann István,Molnár Gergő A

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Endocrinology, Diabetes and Metabolism

Link

https://link.springer.com/content/pdf/10.1186/1475-2840-13-69.pdf

Reference46 articles.

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2. Nyström T, Gutniak MK, Zhang Q, Zhang F, Holst JJ, Ahrén B, Sjöholm A: Effects of glucagon-like-peptide-1 on endothelial function in type 2 diabetes patients with stable coronary artery disease. Am J Physiol Endocrinol Metab. 2004, 287: E1209-E1215. 10.1152/ajpendo.00237.2004.

3. Green BD, Hand KV, Dougan JE, McDonnell BM, Cassidy RS, Grieve DJ: GLP-1 and related peptides cause concentration-dependent relaxation of rat aorta through a pathway involving KATP and cAMP. Arch Biochem Biophys. 2008, 478: 136-142. 10.1016/j.abb.2008.08.001.

4. Nyström T, Gonon AT, Sjöholm A, Pernow J: Glucagon-like peptide-1 relaxes rat conduit arteries via an endothelium-independent mechanism. Regul Pept. 2005, 125: 173-177. 10.1016/j.regpep.2004.08.024.

5. Richter G, Feddersen O, Wagner U, Barth P, Göke B: GLP-1 stimulates secretion of macromolecules from airways and relaxes pulmonary artery. Am J Physiol. 1993, 265: L374-L381.

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