The gut microbiota-derived metabolite trimethylamine N-oxide is elevated in Alzheimer’s disease-Reference-Cited by-同舟云学术

The gut microbiota-derived metabolite trimethylamine N-oxide is elevated in Alzheimer’s disease

Published:2018-12 Issue:1 Volume:10 Page:
ISSN:1758-9193
Container-title:Alzheimer's Research & Therapy
language:en
Short-container-title:Alz Res Therapy

Author:

Vogt Nicholas M.^ORCID,Romano Kymberleigh A.,Darst Burcu F.,Engelman Corinne D.,Johnson Sterling C.,Carlsson Cynthia M.,Asthana Sanjay,Blennow Kaj,Zetterberg Henrik,Bendlin Barbara B.,Rey Federico E.

Funder

National Institute on Aging

National Center for Advancing Translational Sciences

Alzheimerfonden

Torsten Söderbergs Stiftelse

Hjärnfonden

Västra Götalandsregionen

H2020 European Research Council

University of Wisconsin-Madison Microbiome Initiative

Swedish State Support for Clinical Research

Publisher

Springer Science and Business Media LLC

Subject

Cognitive Neuroscience,Clinical Neurology,Neurology

Link

http://link.springer.com/content/pdf/10.1186/s13195-018-0451-2.pdf

Reference42 articles.

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2. Pluznick JL, Protzko RJ, Gevorgyan H, Peterlin Z, Sipos A, Han J, et al. Olfactory receptor responding to gut microbiota-derived signals plays a role in renin secretion and blood pressure regulation. Proc Natl Acad Sci U S A. 2013;110:4410–5.

3. Krautkramer KA, Kreznar JH, Romano KA, Vivas EI, Barrett-Wilt GA, Rabaglia ME, et al. Diet-microbiota interactions mediate global epigenetic programming in multiple host tissues. Mol Cell. 2016;64:982–92.

4. Tremaroli V, Bäckhed F. Functional interactions between the gut microbiota and host metabolism. Nature. 2012;489:242–9.

5. Gao X, Liu X, Xu J, Xue C, Xue Y, Wang Y. Dietary trimethylamine N-oxide exacerbates impaired glucose tolerance in mice fed a high fat diet. J Biosci Bioeng. 2014;118:476–81.

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