The pathophysiological role of acute inflammation after spinal cord injury
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
http://link.springer.com/content/pdf/10.1186/s41232-016-0026-1.pdf
Reference40 articles.
1. Beattie MS. Inflammation and apoptosis: linked therapeutic targets in spinal cord injury. Trends Mol Med. 2004;10:580-3.
2. Ren Y, Young W. Managing inflammation after spinal cord injury through manipulation of macrophage function. Neural Plast. 2013;2013:945034.
3. Okada S, Nakamura M, Fenault-Mihara F, et al. The role of cytokine signaling in pathophysiology for spinal cord injury. Inflamm Regen. 2008;28:440–6.
4. Okada S, Nakamura M, Mikami Y, et al. Blockade of interleukin-6 receptor suppresses reactive astrogliosis and ameliorates functional recovery in experimental spinal cord injury. J Neurosci Res. 2004;76:265–76.
5. Lacroix S, Chang L, Rose-John S, et al. Delivery of hyper-interleukin-6 to the injured spinal cord increases neutrophil and macrophage infiltration and inhibits axonal growth. J Comp Neurol. 2002;454:213–28.
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