SAA1 increases NOX4/ROS production to promote LPS-induced inflammation in vascular smooth muscle cells through activating p38MAPK/NF-κB pathway-Reference-Cited by-同舟云学术

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SAA1 increases NOX4/ROS production to promote LPS-induced inflammation in vascular smooth muscle cells through activating p38MAPK/NF-κB pathway

Published:2019-06-19 Issue:1 Volume:20 Page:
ISSN:2661-8850
Container-title:BMC Molecular and Cell Biology
language:en
Short-container-title:BMC Mol and Cell Biol

Author:

Yu Mei-Hong,Li Xi,Li Qian,Mo Shi-Jing,Ni Yin,Han Fang,Wang Yi-Bin,Tu Yue-Xing

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s12860-019-0197-0.pdf

Reference47 articles.

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2. Yang J, Zhu Y, Lee ET, Zhang Y, Cole SA, Haack K, Best LG, Devereux RB, Roman MJ, Howard BV, et al. Joint associations of 61 genetic variants in the nicotinic acetylcholine receptor genes with subclinical atherosclerosis in American Indians: a gene-family analysis. Circ Cardiovasc Genet. 2013;6(1):89–96.

3. Weber C, Soehnlein O. ApoE controls the interface linking lipids and inflammation in atherosclerosis. J Clin Invest. 2011;121(10):3825–7.

4. Li YT, Swales KE, Thomas GJ, Warner TD, Bishop-Bailey D. Farnesoid x receptor ligands inhibit vascular smooth muscle cell inflammation and migration. Arterioscler Thromb Vasc Biol. 2007;27(12):2606–11.

5. Keramati AR, Singh R, Lin A, Faramarzi S, Ye ZJ, Mane S, Tellides G, Lifton RP, Mani A. Wild-type LRP6 inhibits, whereas atherosclerosis-linked LRP6R611C increases PDGF-dependent vascular smooth muscle cell proliferation. Proc Natl Acad Sci U S A. 2011;108(5):1914–8.

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