The potassium channel KCa3.1 constitutes a pharmacological target for astrogliosis associated with ischemia stroke-Reference-Cited by-同舟云学术

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The potassium channel KCa3.1 constitutes a pharmacological target for astrogliosis associated with ischemia stroke

Published:2017-10-16 Issue:1 Volume:14 Page:
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:J Neuroinflammation

Author:

Yi Mengni,Wei Tianjiao,Wang Yanxia,Lu Qin,Chen Gaoxian,Gao Xiaoling,Geller Herbert M.,Chen Hongzhuan,Yu Zhihua

Funder

Science and Technology Commission of Shanghai Municipality grant

National Natural Science Foundation of China grant

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s12974-017-0973-8.pdf

Reference49 articles.

1. Nedergaard M, Rodriguez JJ, Verkhratsky A. Glial calcium and diseases of the nervous system. Cell Calcium. 2010;47:140–9.

2. Di L, Srivastava S, Zhdanova O, Ding Y, Li Z, Wulff H, Lafaille M, Skolnik EY. Inhibition of the k+ channel kca3.1 ameliorates t cell-mediated colitis. Proc Natl Acad Sci U S A. 2010;107:1541–6.

3. Toyama K, Wulff H, Chandy KG, Azam P, Raman G, Saito T, Fujiwara Y, Mattson DL, Das S, Melvin JE, Pratt PF, Hatoum OA, Gutterman DD, Harder DR, Miura H. The intermediate-conductance calcium-activated potassium channel kca3.1 contributes to atherogenesis in mice and humans. J Clin Invest. 2008;118:3025–37.

4. ZH Y, Wang YX, Song Y, HZ L, Hou LN, Cui YY, Chen HZ. Up-regulation of kca3.1 promotes human airway smooth muscle cell phenotypic modulation. Pharmacol Res. 2013;77:30–8.

5. Bouhy D, Ghasemlou N, Lively S, Redensek A, Rathore KI, Schlichter LC, David S. Inhibition of the ca(2)(+)-dependent k(+) channel, kcnn4/kca3.1, improves tissue protection and locomotor recovery after spinal cord injury. J Neurosci. 2011;31:16298–308.

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