Glial cell activation, recruitment, and survival of B-lineage cells following MCMV brain infection-Reference-Cited by-同舟云学术

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Glial cell activation, recruitment, and survival of B-lineage cells following MCMV brain infection

Published:2016-05-20 Issue:1 Volume:13 Page:
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:J Neuroinflammation

Author:

Lokensgard James R.,Mutnal Manohar B.,Prasad Sujata,Sheng Wen,Hu Shuxian

Funder

National Institute of Neurological Disorders and Stroke

National Institute of Mental Health

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s12974-016-0582-y.pdf

Reference47 articles.

1. Fragkoudis R, Ballany CM, Boyd A, Fazakerley JK. In Semliki Forest virus encephalitis, antibody rapidly clears infectious virus and is required to eliminate viral material from the brain, but is not required to generate lesions of demyelination. J Gen Virol. 2008;89:2565–8.

2. Griffin D, Levine B, Tyor W, Ubol S, Despres P. The role of antibody in recovery from alphavirus encephalitis. Immunol Rev. 1997;159:155–61.

3. Hooper DC, Phares TW, Fabis MJ, Roy A. The production of antibody by invading B cells is required for the clearance of rabies virus from the central nervous system. PLoS Negl Trop Dis. 2009;3:e535.

4. Levine B, Hardwick JM, Trapp BD, Crawford TO, Bollinger RC, Griffin DE. Antibody-mediated clearance of alphavirus infection from neurons. Science. 1991;254:856–60.

5. Stewart BS, Demarest VL, Wong SJ, Green S, Bernard KA. Persistence of virus-specific immune responses in the central nervous system of mice after West Nile virus infection. BMC Immunol. 2011;12:6.

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