CD200R1 agonist attenuates glial activation, inflammatory reactions, and hypersensitivity immediately after its intrathecal application in a rat neuropathic pain model-Reference-Cited by-同舟云学术

CD200R1 agonist attenuates glial activation, inflammatory reactions, and hypersensitivity immediately after its intrathecal application in a rat neuropathic pain model

Published:2016-02-18 Issue:1 Volume:13 Page:
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:J Neuroinflammation

Author:

Hernangómez Miriam,Klusáková Ilona,Joukal Marek,Hradilová-Svíženská Ivana,Guaza Carmen,Dubový Petr

Funder

CEITEC

Employment of Newly Graduated Doctors of Science for Scientific Excellence

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s12974-016-0508-8

Reference74 articles.

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2. Campbell JN, Meyer RA. Mechanisms of neuropathic pain. Neuron. 2006;52(1):77–92.

3. Marchand F, Perretti M, McMahon SB. Role of the immune system in chronic pain. Nature Rev Neurosci. 2005;6(7):521–32.

4. Watkins LR, Hutchinson MR, Ledeboer A, Wieseler-Frank J, Milligan ED, Maier SF. Glia as the “bad guys”: implications for improving clinical pain control and the clinical utility of opioids. Brain Behav Immun. 2007;21(2):131–46.

5. Swett JE, Torigoe Y, Elie VR, Bourassa CM, Miller PG. Sensory neurons of the rat sciatic-nerve. Exp Neurol. 1991;114(1):82–103.

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