Trovafloxacin attenuates neuroinflammation and improves outcome after traumatic brain injury in mice-Reference-Cited by-同舟云学术

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Trovafloxacin attenuates neuroinflammation and improves outcome after traumatic brain injury in mice

Published:2018-02-13 Issue:1 Volume:15 Page:
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:J Neuroinflammation

Author:

Garg Charu,Seo Joon Ho,Ramachandran Jayalakshmi,Loh Ji Meng,Calderon Frances,Contreras Jorge E.

Funder

New Jersey Commission on Brain Injury Research

Health Resources and Services Administration

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s12974-018-1069-9.pdf

Reference81 articles.

1. Gootz TD, Zaniewski R, Haskell S, Schmieder B, Tankovic J, Girard D, Courvalin P, Polzer RJ. Activity of the new fluoroquinolone trovafloxacin (CP-99,219) against DNA gyrase and topoisomerase IV mutants of Streptococcus pneumoniae selected in vitro. Antimicrob Agents Chemother. 1996;40:2691–7.

2. Poon IK, Chiu YH, Armstrong AJ, Kinchen JM, Juncadella IJ, Bayliss DA, Ravichandran KS. Unexpected link between an antibiotic, pannexin channels and apoptosis. Nature. 2014;507:329–34.

3. Chiu YH, Ravichandran KS, Bayliss DA. Intrinsic properties and regulation of pannexin 1 channel. Channels (Austin). 2014;8:103–9.

4. Dahl G. ATP release through pannexon channels. Philos Trans R Soc Lond Ser B Biol Sci. 2015;370

5. Brough D, Pelegrin P, Rothwell NJ. Pannexin-1-dependent caspase-1 activation and secretion of IL-1beta is regulated by zinc. Eur J Immunol. 2009;39:352–8.

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