PTPN22 splice forms: a new role in rheumatoid arthritis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics (clinical),Genetics,Molecular Biology,Molecular Medicine
Reference9 articles.
1. The PTPN22 allele encoding an R620W variant interferes with the removal of developing autoreactive B cells in humans
2. The autoimmune disease–associated PTPN22 variant promotes calpain-mediated Lyp/Pep degradation associated with lymphocyte and dendritic cell hyperresponsiveness
3. Lymphoid tyrosine phosphatase and autoimmunity: human genetics rediscovers tyrosine phosphatases
4. The balance of expression of PTPN22 splice forms is significantly different in rheumatoid arthritis patients compared with controls
5. The PTPN22 R263Q polymorphism is a risk factor for rheumatoid arthritis in Caucasian case-control samples
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