DNA demethylation by 5-aza-2′-deoxycytidine is imprinted, targeted to euchromatin, and has limited transcriptional consequences-Reference-Cited by-同舟云学术

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DNA demethylation by 5-aza-2′-deoxycytidine is imprinted, targeted to euchromatin, and has limited transcriptional consequences

Published:2015-03-17 Issue:1 Volume:8 Page:
ISSN:1756-8935
Container-title:Epigenetics & Chromatin
language:en
Short-container-title:Epigenetics & Chromatin

Author:

Ramos María-Paz,Wijetunga Neil Ari,McLellan Andrew S,Suzuki Masako,Greally John M

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13072-015-0004-x.pdf

Reference46 articles.

1. Mund C, Lyko F. Epigenetic cancer therapy: proof of concept and remaining challenges. Bioessays. 2010;32:949–57.

2. Wiech NL, Fisher JF, Helquist P, Wiest O. Inhibition of histone deacetylases: a pharmacological approach to the treatment of non-cancer disorders. Curr Top Med Chem. 2009;9:257–71.

3. Chuang JC, Yoo CB, Kwan JM, Li TW, Liang G, Yang AS, et al. Comparison of biological effects of non-nucleoside DNA methylation inhibitors versus 5-aza-2′-deoxycytidine. Mol Cancer Ther. 2005;4:1515–20.

4. Stresemann C, Brueckner B, Musch T, Stopper H, Lyko F. Functional diversity of DNA methyltransferase inhibitors in human cancer cell lines. Cancer Res. 2006;66:2794–800.

5. Ghoshal K, Datta J, Majumder S, Bai S, Kutay H, Motiwala T, et al. 5-Aza-deoxycytidine induces selective degradation of DNA methyltransferase 1 by a proteasomal pathway that requires the KEN box, bromo-adjacent homology domain, and nuclear localization signal. Mol Cell Biol. 2005;25:4727–41.

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