Cells adapt to the epigenomic disruption caused by histone deacetylase inhibitors through a coordinated, chromatin-mediated transcriptional response
Author:
Funder
Cancer Research UK
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology
Link
http://link.springer.com/content/pdf/10.1186/s13072-015-0021-9
Reference89 articles.
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2. Marmorstein R, Trievel RC. Histone modifying enzymes: structures, mechanisms, and specificities. Biochim Biophys Acta. 2009;1789:58–68.
3. Covault J, Chalkley R. The identification of distinct populations of acetylated histone. J Biol Chem. 1980;255:9110–6.
4. Nightingale KP, Gendreizig S, White DA, Bradbury C, Hollfelder F, Turner BM. Cross-talk between histone modifications in response to histone deacetylase inhibitors: MLL4 links histone H3 acetylation and histone H3K4 methylation. J Biol Chem. 2007;282:4408–16.
5. Ekwall K. Genome-wide analysis of HDAC function. Trends Genet. 2005;21:608–15.
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