The transcription factor Foxg1 regulates telencephalic progenitor proliferation cell autonomously, in part by controlling Pax6 expression levels-Reference-Cited by-同舟云学术

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The transcription factor Foxg1 regulates telencephalic progenitor proliferation cell autonomously, in part by controlling Pax6 expression levels

Published:2011 Issue:1 Volume:6 Page:9
ISSN:1749-8104
Container-title:Neural Development
language:en
Short-container-title:Neural Dev

Author:

Manuel Martine N,Martynoga Ben,Molinek Mike D,Quinn Jane C,Kroemmer Corinne,Mason John O,Price David J

Publisher

Springer Science and Business Media LLC

Subject

Developmental Neuroscience

Reference24 articles.

1. Pax6 controls cerebral cortical cell number by regulating exit from the cell cycle and specifies cortical cell identity by a cell autonomous mechanism

2. Controlled overexpression of Pax6 in vivo negatively autoregulates the Pax6 locus, causing cell-autonomous defects of late cortical progenitor proliferation with little effect on cortical arealization

3. Winged helix transcription factor BF-1 is essential for the development of the cerebral hemispheres

4. Foxg1 is required for specification of ventral telencephalon and region-specific regulation of dorsal telencephalic precursor proliferation and apoptosis

5. Role of Pax6 in forebrain regionalization

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