PAT1 inversely regulates the surface Amyloid Precursor Protein level in mouse primary neurons-Reference-Cited by-同舟云学术

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PAT1 inversely regulates the surface Amyloid Precursor Protein level in mouse primary neurons

Published:2015-03-07 Issue:1 Volume:16 Page:
ISSN:1471-2202
Container-title:BMC Neuroscience
language:en
Short-container-title:BMC Neurosci

Author:

Dilsizoglu Senol Aysegul,Tagliafierro Lidia,Huguet Léa,Gorisse-Hussonnois Lucie,Chasseigneaux Stéphanie,Allinquant Bernadette

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Neuroscience

Link

http://link.springer.com/article/10.1186/s12868-015-0152-8/fulltext.html

Reference44 articles.

1. Selkoe DJ. Alzheimer’s disease: genes, proteins, and therapy. Physiol Rev. 2001;81(2):741–66.

2. Gandy S. The role of cerebral amyloid beta accumulation in common forms of Alzheimer disease. J Clin Invest. 2005;115(5):1121–9.

3. Brouillet E, Trembleau A, Galanaud D, Volovitch M, Bouillot C, Valenza C, et al. The amyloid precursor protein interacts with Go heterotrimeric protein within a cell compartment specialized in signal transduction. J Neurosci. 1999;19(5):1717–27.

4. Torroja L, Packard M, Gorczyca M, White K, Budnik V. The Drosophila beta-amyloid precursor protein homolog promotes synapse differentiation at the neuromuscular junction. J Neurosci. 1999;19(18):7793–803.

5. Rohn TT, Ivins KJ, Bahr BA, Cotman CW, Cribbs DH. A monoclonal antibody to amyloid precursor protein induces neuronal apoptosis. J Neurochem. 2000;74(6):2331–42.

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4. Protein interacting with Amyloid Precursor Protein tail-1 (PAT1) is involved in early endocytosis;Cellular and Molecular Life Sciences;2019-05-28

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