Re-engineering a neuroprotective, clinical drug as a procognitive agent with high in vivo potency and with GABAA potentiating activity for use in dementia-Reference-Cited by-同舟云学术

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Re-engineering a neuroprotective, clinical drug as a procognitive agent with high in vivo potency and with GABAA potentiating activity for use in dementia

Published:2015-10-19 Issue:1 Volume:16 Page:
ISSN:1471-2202
Container-title:BMC Neuroscience
language:en
Short-container-title:BMC Neurosci

Author:

Luo Jia,Lee Sue H.,VandeVrede Lawren,Qin Zhihui,Piyankarage Sujeewa,Tavassoli Ehsan,Asghodom Rezene T.,Aissa Manel Ben,Fà Mauro,Arancio Ottavio,Yue Lan,Pepperberg David R.,Thatcher Gregory R. J.

Funder

NIH Clinical Center

Alzheimer's Drug Discovery Foundation

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s12868-015-0208-9.pdf

Reference85 articles.

1. Thies W, Bleiler L. 2013 Alzheimer’s disease facts and figures. Alzheimers Dement. 2013;9(2):208–45.

2. Tanzi RE, Kovacs DM, Kim TW, Moir RD, Guenette SY, Wasco W. The gene defects responsible for familial Alzheimer’s disease. Neurobiol Dis. 1996;3(3):159–68.

3. Lue LF, Kuo YM, Roher AE, Brachova L, Shen Y, Sue L, Beach T, Kurth JH, Rydel RE, Rogers J. Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer’s disease. Am J Pathol. 1999;155(3):853–62.

4. McLean CA, Cherny RA, Fraser FW, Fuller SJ, Smith MJ, Beyreuther K, Bush AI, Masters CL. Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer’s disease. Ann Neurol. 1999;46(6):860–6.

5. Wang J, Dickson DW, Trojanowski JQ, Lee VM. The levels of soluble versus insoluble brain Abeta distinguish Alzheimer’s disease from normal and pathologic aging. Exp Neurol. 1999;158(2):328–37.

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