Genetic deletion of calcium/calmodulin-dependent protein kinase kinase β (CaMKK β) or CaMK IV exacerbates stroke outcomes in ovariectomized (OVXed) female mice-Reference-Cited by-同舟云学术

Genetic deletion of calcium/calmodulin-dependent protein kinase kinase β (CaMKK β) or CaMK IV exacerbates stroke outcomes in ovariectomized (OVXed) female mice

Published:2014-10-21 Issue:1 Volume:15 Page:
ISSN:1471-2202
Container-title:BMC Neuroscience
language:en
Short-container-title:BMC Neurosci

Author:

Liu Lin,McCullough Louise,Li Jun

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Neuroscience

Link

http://link.springer.com/article/10.1186/s12868-014-0118-2/fulltext.html

Reference28 articles.

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2. Del Zoppo GJ, Saver JL, Jauch EC, Adams HP: Expansion of the time window for treatment of acute ischemic stroke with intravenous tissue plasminogen activator a science advisory from the American Heart Association/American Stroke Association. Stroke. 2009, 40: 2945-2948. 10.1161/STROKEAHA.109.192535.

3. Manwani B, McCullough LD: Sexual dimorphism in ischemic stroke: lessons from the laboratory. Womens Health (Lond Engl). 2011, 7: 319-339. 10.2217/whe.11.22.

4. Li J, Benashski SE, Siegel C, Liu F, McCullough LD: Adenosine monophosphate activated protein kinase inhibition is protective in both sexes after experimental stroke. Neurosci Lett. 2010, 482: 62-65. 10.1016/j.neulet.2010.07.007.

5. Yuan M, Siegel C, Zeng Z, Li J, Liu F, McCullough LD: Sex differences in the response to activation of the poly (ADP-ribose) polymerase pathway after experimental stroke. Exp Neurol. 2009, 217: 210-218. 10.1016/j.expneurol.2009.02.012.

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