HDAC class I inhibitor, Mocetinostat, reverses cardiac fibrosis in heart failure and diminishes CD90+ cardiac myofibroblast activation-Reference-Cited by-同舟云学术

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HDAC class I inhibitor, Mocetinostat, reverses cardiac fibrosis in heart failure and diminishes CD90+ cardiac myofibroblast activation

Published:2014-07-02 Issue:1 Volume:7 Page:
ISSN:1755-1536
Container-title:Fibrogenesis & Tissue Repair
language:en
Short-container-title:Fibrogenesis Tissue Repair

Author:

Nural-Guvener Hikmet F,Zakharova Luidmila,Nimlos James,Popovic Snjezana,Mastroeni Diego,Gaballa Mohamed A

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology,Dermatology,Hepatology,Rheumatology,Medicine (miscellaneous)

Link

https://link.springer.com/content/pdf/10.1186/1755-1536-7-10.pdf

Reference39 articles.

1. Porter KE, Turner NA: Cardiac fibroblasts: at the heart of myocardial remodeling. Pharmacol Ther. 2009, 132: 255-278.

2. Squires CE, Escobar GP, Payne JF, Leonardi RA, Goshorn DK, Sheats NJ, Mains IM, Mingoia JT, Flack EC, Lindsey ML: Altered fibroblast function following myocardial infarction. J Mol Cell Cardiol. 2005, 39: 699-707.

3. van den Borne SW, Diez J, Blankesteijn WM, Verjans J, Hofstra L, Narula J: Myocardial remodeling after infarction: the role of myofibroblasts. Nat Rev Cardiol. 2010, 7: 30-37.

4. Xie M, Hill JA: HDAC-dependent ventricular remodeling. Trends Cardiovasc Med. 2013, 23: 229-235.

5. Montgomery RL, Davis CA, Potthoff MJ, Haberland M, Fielitz J, Qi X, Hill JA, Richardson JA, Olson EN: Histone deacetylases 1 and 2 redundantly regulate cardiac morphogenesis, growth, and contractility. Genes Dev. 2007, 21: 1790-1802.

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