MEF2A alters the proliferation, inflammation-related gene expression profiles and its silencing induces cellular senescence in human coronary endothelial cells-Reference-Cited by-同舟云学术

MEF2A alters the proliferation, inflammation-related gene expression profiles and its silencing induces cellular senescence in human coronary endothelial cells

Published:2019-03-18 Issue:1 Volume:20 Page:
ISSN:1471-2199
Container-title:BMC Molecular Biology
language:en
Short-container-title:BMC Molecular Biol

Author:

Xiong Yujuan,Wang Lin,Jiang Wenyi,Pang Lihua,Liu Weihua,Li Aiqun,Zhong Yun,Ou Wenchao,Liu Benrong,Liu Shi-ming

Funder

the innovation project from the Department of Education of Guangdong Province

Guangdong Provincial Science and Technology plan project

the General Programs of the National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s12867-019-0125-z.pdf

Reference40 articles.

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3. Kaushal S, Schneider JW, Nadal-Ginard B, Mahdavi V. Activation of the myogenic lineage by MEF2A, a factor that induces and cooperates with MyoD. Science. 1994;266(5188):1236–40.

4. Erck C, Seidl K. MyoD and MEF2A mediate activation and repression of the p75NGFR gene during muscle development. Biochem Biophys Res Commun. 1998;245(3):871–7.

5. Naya FJ, Black BL, Wu H, Bassel-Duby R, Richardson JA, Hill JA, Olson EN. Mitochondrial deficiency and cardiac sudden death in mice lacking the MEF2A transcription factor. Nat Med. 2002;8(11):1303–9.

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