The Genetic Analysis Workshop 16 Problem 3: simulation of heritable longitudinal cardiovascular phenotypes based on actual genome-wide single-nucleotide polymorphisms in the Framingham Heart Study-Reference-Cited by-同舟云学术

The Genetic Analysis Workshop 16 Problem 3: simulation of heritable longitudinal cardiovascular phenotypes based on actual genome-wide single-nucleotide polymorphisms in the Framingham Heart Study

Published:2009-12 Issue:S7 Volume:3 Page:
ISSN:1753-6561
Container-title:BMC Proceedings
language:en
Short-container-title:BMC Proc

Author:

Kraja Aldi T,Culverhouse Robert,Daw E Warwick,Wu Jun,Van Brunt Andrew,Province Michael A,Borecki Ingrid B

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1186/1753-6561-3-S7-S4.pdf

Reference9 articles.

1. Kannel WB, Dawber TR, Kagan A, Revotskie N, Stokes J: Factors of risk in the development of coronary heart disease--six year follow-up experience. The Framingham Study. Ann Intern Med. 1961, 55: 33-50.

2. Kathiresan S, Manning AK, Demissie S, D'Agostino RB, Surti A, Guiducci C, Gianniny L, Burtt NP, Melander O, Orho-Melander M, Arnett DK, Peloso GM, Ordovas JM, Cupples LA: A genome-wide association study for blood lipid phenotypes in the Framingham Heart Study. BMC Med Genet. 2007, 8 (suppl 1): S17-10.1186/1471-2350-8-S1-S17.

3. Kraja AT, Rao DC, Weder AB, Cooper R, Curb JD, Hanis CL, Turner ST, de Andrade M, Hsiung CA, Quertermous T, Zhu X, Province MA: Two major QTLs and several others relate to factors of metabolic syndrome in the family blood pressure program. Hypertension. 2005, 46: 751-757. 10.1161/01.HYP.0000184249.20016.bb.

4. Kannel WB: Risk stratification of dyslipidemia: insights from the Framingham Study. Curr Met Chem Cardiovasc Hematol Agents. 2005, 3: 187-193. 10.2174/1568016054368250.

5. Almasy L, Terwilliger JD, Nielsen D, Dyer TD, Zaykin D, Blangero J: GAW12: simulated genome scan, sequence, and family data for a common disease. Genet Epidemiol. 2001, 21 (suppl 1): S332-S338.

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