Mitochondrial dysfunction in down syndrome: molecular mechanisms and therapeutic targets-Reference-Cited by-同舟云学术

Mitochondrial dysfunction in down syndrome: molecular mechanisms and therapeutic targets

Published:2018-03-15 Issue:1 Volume:24 Page:
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

Izzo Antonella,Mollo Nunzia,Nitti Maria,Paladino Simona,Calì Gaetano,Genesio Rita,Bonfiglio Ferdinando,Cicatiello Rita,Barbato Maria,Sarnataro Viviana,Conti Anna^ORCID,Nitsch Lucio

Funder

Regione Campania

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1186/s10020-018-0004-y.pdf

Reference81 articles.

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3. Arbuzova S, Hutchin T, Cuckle H. Mitochondrial dysfunction and Down's syndrome. BioEssays. 2002;24:681–4.

4. Arron JR, Winslow MM, Polleri A, Chang CP, Wu H, Gao X, et al. NFAT dysregulation by increased dosage of DSCR1 and DYRK1A on chromosome 21. Nature. 2006;441:595–600.

5. Bartley MG, Marquardt K, Kirchhof D, Wilkins HM, Patterson D, Linseman DA. Overexpression of amyloid-beta protein precursor induces mitochondrial oxidative stress and activates the intrinsic apoptotic cascade. J Alzheimers Dis. 2012;28:855–68.

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