Uncoupling of glycolysis from glucose oxidation accompanies the development of heart failure with preserved ejection fraction-Reference-Cited by-同舟云学术

Uncoupling of glycolysis from glucose oxidation accompanies the development of heart failure with preserved ejection fraction

Published:2018-03-15 Issue:1 Volume:24 Page:
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

Fillmore Natasha,Levasseur Jody L.,Fukushima Arata,Wagg Cory S.,Wang Wei,Dyck Jason R. B.,Lopaschuk Gary D.

Funder

Canadian Institutes of Health Research

Alberta Innovates - Health Solutions

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1186/s10020-018-0005-x.pdf

Reference54 articles.

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2. Barr RL, Lopaschuk GD. Direct measurement of energy metabolism in the isolated working rat heart. J Pharmacol Toxicol Methods. 1997;38:11–7.

3. Beer M, Seyfarth T, Sandstede J, Landschutz W, Lipke C, Kostler H, et al. Absolute concentrations of high-energy phosphate metabolites in normal, hypertrophied, and failing human myocardium measured noninvasively with (31)P-SLOOP magnetic resonance spectroscopy. J Am Coll Cardiol. 2002;40:1267–74.

4. Bonnet S, Archer SL, Allalunis-Turner J, Haromy A, Beaulieu C, Thompson R, et al. A mitochondria-K+ channel axis is suppressed in cancer and its normalization promotes apoptosis and inhibits cancer growth. Cancer Cell. 2007;11:37–51.

5. Canadian Council on Animal Care. (2017) Guide to the care and use of experimental animals. 2nd. Ottawa, Ontario. [cited 2017 Sept 16]. Available from: http://www.ccac.ca/ .

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