A resting EEG study of neocortical hyperexcitability and altered functional connectivity in fragile X syndrome-Reference-Cited by-同舟云学术

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A resting EEG study of neocortical hyperexcitability and altered functional connectivity in fragile X syndrome

Published:2017-03-14 Issue:1 Volume:9 Page:
ISSN:1866-1947
Container-title:Journal of Neurodevelopmental Disorders
language:en
Short-container-title:J Neurodevelop Disord

Author:

Wang Jun,Ethridge Lauren E.,Mosconi Matthew W.,White Stormi P.,Binder Devin K.,Pedapati Ernest V.,Erickson Craig A.,Byerly Matthew J.,Sweeney John A.

Funder

National Institute of Child Health and Human Development

Publisher

Springer Science and Business Media LLC

Subject

Cognitive Neuroscience,Neurology (clinical),Pathology and Forensic Medicine,Pediatrics, Perinatology and Child Health

Link

http://link.springer.com/content/pdf/10.1186/s11689-017-9191-z.pdf

Reference60 articles.

1. Ashley CJ, Wilkinson K, Reines D, Warren S. FMR1 protein: conserved RNP family domains and selective RNA binding. Science. 1993;262:563–6.

2. Bear MF. Therapeutic implications of the mGluR theory of fragile X mental retardation. Genes Brain Behav. 2005;4(6):393–8.

3. Heulens I, D'Hulst C, Braat S, Rooms L, Kooy RF. Involvement and therapeutic potential of the GABAergic system in the fragile x syndrome. Sci World J. 2010;10:2198–206.

4. Cea-Del Rio CA, Huntsman MM. The contribution of inhibitory interneurons to circuit dysfunction in Fragile X Syndrome. Front Cell Neurosci. 2014;8:1–7.

5. Contractor A, Klyachko VA, Portera-Cailliau C. Altered neuronal and circuit excitability in fragile X syndrome. Neuron. 2015;87(4):699–715.

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