Persistent motor dysfunction despite homeostatic rescue of cerebellar morphogenesis in the Car8 waddles mutant mouse-Reference-Cited by-同舟云学术

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Persistent motor dysfunction despite homeostatic rescue of cerebellar morphogenesis in the Car8 waddles mutant mouse

Published:2019-03-12 Issue:1 Volume:14 Page:
ISSN:1749-8104
Container-title:Neural Development
language:en
Short-container-title:Neural Dev

Author:

Miterko Lauren N.,White Joshua J.,Lin Tao,Brown Amanda M.,O’Donovan Kevin J.,Sillitoe Roy V.

Funder

Eunice Kennedy Shriver National Institute of Child Health and Human Development

National Institutes of Neurological Disorders and Stroke

The Hamill Foundation

Publisher

Springer Science and Business Media LLC

Subject

Developmental Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s13064-019-0130-4.pdf

Reference106 articles.

1. Orr HT. SCA1-phosphorylation, a regulator of Ataxin-1 function and pathogenesis. Prog Neurobiol. 2012;99:179–85.

2. Ledoux MS, Lorden JF. Abnormal spontaneous and harmaline-stimulated Purkinje cell activity in the awake genetically dystonic rat. Exp Brain Res. 2002;145:457–67.

3. Wilson BK, Hess EJ. Animal models for dystonia. Mov Disord. 2013;28:982–9.

4. Louis ED, Faust PL, J-PG V. Purkinje cell loss is a characteristic of essential tremor. Park Relat Disord. 2011;17:406–9.

5. Gennarino VA, Singh RK, White JJ, De Maio A, Han K, Kim JY, et al. Pumilio1 haploinsufficiency leads to SCA1-like neurodegeneration by increasing wild-type Ataxin1 levels. Cell. 2015;160:1087–98.

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