The histone deacetylase inhibitor trichostatin a decreases lymphangiogenesis by inducing apoptosis and cell cycle arrest via p21-dependent pathways-Reference-Cited by-同舟云学术

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The histone deacetylase inhibitor trichostatin a decreases lymphangiogenesis by inducing apoptosis and cell cycle arrest via p21-dependent pathways

Published:2016-09-30 Issue:1 Volume:16 Page:
ISSN:1471-2407
Container-title:BMC Cancer
language:en
Short-container-title:BMC Cancer

Author:

Hrgovic Igor,Doll Monika,Kleemann Johannes,Wang Xiao-Fan,Zoeller Nadja,Pinter Andreas,Kippenberger Stefan,Kaufmann Roland,Meissner Markus

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Oncology

Link

http://link.springer.com/content/pdf/10.1186/s12885-016-2807-y.pdf

Reference76 articles.

1. de Ruijter AJ, van Gennip AH, Caron HN, Kemp S, van Kuilenburg AB. Histone deacetylases (HDACs). Characterization of the classical HDAC family. Biochem J. 2003;370:737–49.

2. Villar-Garea A, Esteller M. Histone deacetylase inhibitors: understanding a new wave of anticancer agents. Int J Cancer. 2004;112:171–8.

3. Yang XJ, Seto E. HATs and HDACs. From structure, function and regulation to novel strategies for therapy and prevention. Oncogene. 2007;26:5310–8.

4. West AC, Johnstone RW. New and emerging HDAC inhibitors for cancer treatment. J Clin Invest. 2014;124:30–9.

5. Li Z, Zhu WG. Targeting histone deacetylases for cancer therapy: from molecular mechanisms to clinical implications. Int J Biol Sci. 2014;10:757–70.

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