Sertad1 antagonizes iASPP function by hindering its entrance into nuclei to interact with P53 in leukemic cells-Reference-Cited by-同舟云学术

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Sertad1 antagonizes iASPP function by hindering its entrance into nuclei to interact with P53 in leukemic cells

Published:2017-11-28 Issue:1 Volume:17 Page:
ISSN:1471-2407
Container-title:BMC Cancer
language:en
Short-container-title:BMC Cancer

Author:

Qiu Shaowei,Liu Shuang,Yu Tengteng,Yu Jing,Wang Min,Rao Qing,Xing Haiyan,Tang Kejing,Mi Yinchang,Wang Jianxiang

Funder

National Natural Science Foundation of China

Tianjin Major Science and Technology Project

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Oncology

Link

http://link.springer.com/content/pdf/10.1186/s12885-017-3787-2.pdf

Reference31 articles.

1. Levine AJ, Oren M. The first 30 years of p53: growing ever more complex. Nature Rev Cancer. 2009;9:749–58.

2. Boyapati A, Kanbe E. Zhang DE.P53 alterations in myeloid leukemia. Acta Haematol. 2004;111:100–6.

3. Ok CY, Patel KP, Garcia-Manero G, Routbort MJ, Peng J, Tang G, et al. TP53 mutation characteristics in therapy-related myelodysplastic syndromes and acute myeloid leukemia issimilar to de novo diseases. J Hematol Oncol. 2015;8:45.

4. Saha MN, Qiu L, Chang H. Targeting p53 by small molecules in hematological malignanies. J Hematol Oncol. 2013;6:23.

5. Yang JP, Hori M, Takahashi N, Kawabe T, Kato H, Okamoto T. NF-kappaB subunit p65 binds to 53BP2 and inhibits cell death induced by 53BP2. Oncogene. 1999;18:5177–86.

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